Here’s how California could be missing pesticides’ cancer risk – #sayNOtoPESTICIDES!

Strawberry_Fields.jpg

The local community is concerned about high levels of pesticides used near Rio Mesa High School in Oxnard, Calif., which is surrounded by strawberry fields on all four sides.  Photo:Sam Hodgson

February 17, 2016 | by Andrew Donohue | Reveal

The local community is concerned about high levels of pesticides used near Rio Mesa High School in Oxnard, Calif., which is surrounded by strawberry fields on all four sides.

California’s pesticide police could be missing a serious health concern for residents and farmworkers by failing to monitor what happens when pesticides get mixed together.

As a new report from UCLA highlighted today, California studies only how each individual pesticide affects human health. Often, however, workers and residents are exposed to a number of pesticides at the same time.

That can happen when pesticides get mixed together before they’re applied to fields or when different pesticides are used in the same field on the same day. A growing body of science is showing that the chemical cocktails could create greater health risks than each pesticide does on its own.

In particular, the report shows how three fumigants – a type of gaseous pesticide central to the strawberry industry and used near schools and homes – might combine to increase the risk of cancer for bystanders. Essentially, once in the human body together, the chemicals can team up to attack and mutate DNA in a way they wouldn’t on their own.

“The regulatory system that is supposed to protect people from harmful levels of pesticide exposure has been slow to deal with interactive effects when setting exposure limits for pesticides,” the report says.

The California Department of Pesticide Regulation’s mission is to protect humans and the environment from the dangers of pesticides. The report’s authors, who come from UCLA’s law and public health schools, said the department must begin studying the combined effects. And they point out that low-income and minority residents are at the greatest risk.

“DPR is required to assess this risk and protect public health, but isn’t doing so,” the authors wrote.

The department already is under fire for how it has managed fumigants, which can spread easily through the air. A Reveal investigation found that department leaders allowed growers and Dow AgroSciences to use heavy amounts of one fumigant despite strenuous objections of scientists because of its potential to cause cancer.

When Ventura County residents subsequently raised concern about the pesticide’s use in strawberry fields near Rio Mesa High School, department Director Brian Leahy responded with a series of exaggerations and contradictions.

The department has curtailed the pesticide’s use and begun drafting rules that would limit pesticide use around schools and require residents to be notified of fumigant use near their homes. However, the state continues to keep open the loophole it created at Dow’s request.

Last week, the department’s second-in-charge, Chris Reardon, left without explanation after nearly 13 years with the agency. An appointee of the governor, Reardon maintained close ties with the agricultural industry, copies of his calendars show.

The UCLA report focused on the fields around Rio Mesa High School to make its case. The school is boxed in on all four sides by conventional strawberry fields. Although pesticides aren’t applied during school hours, the gases can linger in the air for weeks after they’re applied without teachers or students knowing.

Combined, the health risk could be much greater than those of the individual pesticides.

“In fact, modeling shows that over the course of about one week people who live and work in the area around Rio Mesa High School in Ventura County were exposed to large doses of multiple fumigants,” the report says. “This level of exposure raises concerns about possible interactive effects.”

The report points out that 35 percent of all fumigants were applied on the same field on the same day as another fumigant, and 26 percent were applied as part of a pesticide mix.

The authors recommend the following changes in California’s pesticide regulation:

  • Pesticides sold as part of a mixture should be tested before being approved for use.
  • When pesticides are mixed at the field or applied near each other, regulators should require testing or create strict restrictions if there’s a reasonable chance of human harm.
  • The combined effects of the pesticides should be considered in the initial health research done by the Department of Pesticide Regulation and the rules it creates around the pesticides’ use.

#SayNOtoPESTICIDES!

BedBugs reported in some of NYC’s swankiest hotels. They were always there; and it’s getting worse. More important to follow as BedBugs transmit deadly Chagas disease.

February 8, 2016 | by Leonard Greene | New York Daily News
It’s not just the fleabags and flophouses.

Bedbugs have been reported in some of the city’s swankiest hotels with a list that includes the Waldorf Astoria the Millennium Hilton and the New York Marriott Marquis.

According to the Bedbug Registry, a nationwide database of bedbug reports and complaints, bedbug sightings in New York hotels have jumped more than 44 percent between 2014 and 2015.

The Millenium Hilton at 55 Church Street in New York New York.
Google Maps Street View

The Millenium Hilton at 55 Church Street in New York New York.

The data focused on establishments that are members of the Hotel Association of New York City.

Of the 272 association members, 65 percent, or 176 members, have had a guest file at least one complaint about bedbugs at the property.

NR

Michelle Bennett/Getty Images/Lonely Planet Image

Taxi cabs outside Waldorf Astoria Hotel.

Eighteen hotels had a combined 363 complaints, representing 42 percent of all bedbug complaints.

“I stayed in room 2306 for one night,” a Millennium Hilton guest wrote in a complaint to the hotel in 2014. “I found blood on my sheets and a live bug on my bed. I ended up with 60 plus bites.”

At the Times Square Doubletree guest said a stay there last year left hundreds of bite marks on the face, neck arms and hands.

“Extreme case of bed bug attacked on my entire upper body,” the guest wrote.” Went home to Florida a day early and ended up in my local emergency room.”

Research Entomologist Jeffrey White shows off some bedbugs at a informational bedbug conference at 201 Mulberry Street in Manhattan Wednesday.

Warga, Craig/New York Daily News

Last month, a California couple posted a YouTube video about their $400-a-night Central Park hotel room nightmare. The couple found dozens of bedbugs beneath their mattress at the Astor on the Park Hotel.

Lisa Linden, a spokeswoman for the hotel association, said hotels in New York are addressing the issue.

“Bedbugs are a global issue that extend beyond hotels,” Linden said.

”Every member of the Hotel Association of NYC that we are aware of has an active anti-bedbug program in place. If a problem arises, it is dealt with immediately and effectively.”

Scientists who recently studied the bloodsucking creatures in the city’s subway system discovered a genetic diversity among bedbugs depending upon the neighborhood where they were found.

They said the discovery could lead to better insecticides.

#SayNOtoPESTICIDES!

Watch this BedBug go from flat to FAT

FYI – A 2014 Penn State Study confirms that Chagas CAN be transmitted by bed bugs and that bed bugs carry 40 other pathogens and MRSA.  ~A. Steiner~

February 8, 2016 | Christopher Terrell Nield | The Conversation
The bedbug problem is getting worse. Infestation horror stories have popped up in most major cities and a pest control team was even asked recently to exterminate bedbugs on an offshore oil rig.

We tend to associate bedbugs with dirty living conditions, but this is a myth – they don’t actually choose dirty homes over clean ones.  Unusually for many blood-sucking insects, bed bugs haven’t (yet) been implicated in spreading disease to the humans they bite, so that’s one small thing in their favour, though they are suspected of carrying organisms that cause leprosy, oriental sore and the bacterial brucellosis, and may be able to transmit Chagas disease.

After feasting on human blood the same bedbug goes from flat (00:25) to fat (3:25).

#SayNOtoPESTICIDES

Genome of BedBug shows close relationships to Kissing Bug, one of several vectors for deadly Chagas disease, and the body louse. Both have tight associations with humans.

February 2, 2016 | News from Weill Cornell Medical College

Researchers Sequence First Bedbug Genome.  Scientists have assembled the first complete genome of one of humanity’s oldest and least-loved companions: the bedbug. The new work, led by researchers at the American Museum of Natural History and Weill Cornell Medicine, and published Feb. 2 in Nature Communications, could help combat pesticide resistance in the unwelcome parasite. The data also provides a rich genetic resource for mapping bedbug activity in human hosts and in cities, including subways.

male and female bedbugs – both fed and unfed – comparison with apple seeds

“Bedbugs are one of New York City’s most iconic living fossils, along with cockroaches, meaning that their outward appearance has hardly changed throughout their long lineage,” said one of the paper’s corresponding authors Dr. George Amato, director of the museum’s Sackler Institute for Comparative Genomics. “But despite their static look, we know that they continue to evolve, mostly in ways that make it harder for humans to dissociate with them. This work gives us the genetic basis to explore the bedbug’s basic biology and its adaptation to dense human environments.”

The common bedbug (Cimex lectularius) has been coupled with humans for thousands of years. This species is found in temperate regions and prefers to feed on human blood. In recent decades, the prevalence of heated homes and global air travel has accelerated infestations in urban areas, where bedbugs have constant access to blood meals and opportunities to migrate to new hosts. A resurgence in bedbug infestations since the late 1990s is largely associated with the evolution of the insects’ resistance to known pesticides, many of which are not suitable for indoor application.

“Bedbugs all but vanished from human lives in the 1940s because of the widespread use of DDT, but unfortunately, overuse contributed to resistance issues quite soon after that in bedbugs and other insect pests,” said Louis Sorkin, an author on the paper and a senior scientific assistant in the Museum’s Division of Invertebrate Zoology. “Today, a very high percentage of bedbugs have genetic mutations that make them resistant to the insecticides that were commonly used to battle these urban pests. This makes the control of bedbugs extremely labor intensive.”

The researchers extracted DNA and RNA from preserved and living collections, including samples from a population that was first collected in 1973 and has been maintained by museum staff members since then. RNA was sampled from males and females representing each of the bug’s six life stages, before and after blood meals, in order to paint a full picture of the bedbug genome.

When compared with 20 other arthropod genomes, the genome of the common bedbug shows close relationships to the kissing bug (Rhodnius prolixus), one of several vectors for Chagas disease, and the body louse (Pediculus humanus), which both have tight associations with humans.

Click here to read complete article.

 

SayNOtoPESTICIDES!

Bed Bugs show resistance to pesticides: What to do now? Build a wall!

Why chemicals used to fight bed bugs aren’t working any longer was revealed in a new study that compared today’s bed bugs with those that have been isolated in a lab for 30 years.

February 1, 2016 | by Lonnie Shekhtman  | The Christian Science Monitor

The chemicals used to fight bed bug infestations are no longer working, say scientists from Virginia Polytechnic Institute and New Mexico State University. The tiny pests have developed a resistance to the most commonly used type of insecticides, called neonicotinoids, or neonics, which is part of the reason there has been a resurgence of them in the last couple of decades.

“While we all want a powerful tool to fight bed bug infestations, what we are using as a chemical intervention is not working as effectively it was designed and, in turn, people are spending a lot of money on products that aren’t working,” Troy Anderson, an assistant professor of entomology in the Virginia Tech College of Agriculture and Life Sciences, said in an announcement last week.

In an experiment, researchers compared bed bugs from homes in Cincinnati and Michigan that had been previously exposed to neonics with those that a researcher has kept isolated in a lab for 30 years, dating back to a time before the insecticides were used commercially.

In results published Thursday in the Journal of Medical Entomology, Dr. Anderson and Alvaro Romero, an assistant professor of entomology at New Mexico State University, reported that the bed bugs that had been isolated in a lab for 30 years died when treated with a small amount of neonics. Those collected from homes in Cincinnati and Michigan showed much higher resistance to the chemical treatment.

The team also tested bedbugs from New Jersey that were already resistant to pyrethroids, another class of widely used insecticides often mixed with neonics, but had been isolated from neonics since 2008. Those bugs were more susceptible to the insecticides than the ones from Cincinnati and Michigan, but not as much as the isolated bedbugs.

“Companies need to be vigilant for hints of declining performance of products that contain neonicotinoids,” Dr. Romero said in a study announcement.

“For example, bed bugs persisting on previously treated surfaces might be an indication of resistance. In these cases, laboratory confirmation of resistance is advised, and if resistance is detected, products with different modes of action need to be considered, along with the use of non-chemical methods,” he said.

Bed bugs are particularly burdensome in apartment buildings, where they can spread to many units. They are also more problematic for low-income, elderly, and disabled people who can’t spot the tiny red bug and often don’t have the means to get rid of them, say researchers from Virginia Tech.

Bed bugs thrive in beds, couches, and around electrical outlets and cause hundreds of bites a night.

“When well-off people get bed bugs, it’s an inconvenience. But when low-income families get them, there aren’t many options,” said Molly Stedfast, who worked with bed bugs as a graduate entomology student at Virginia Tech in 2013.

“Those who can’t afford the treatments,” she says, often end up living with bed bugs for a long time.

Virginia Tech’s pest lab recommends a nontoxic, non-neonic treatment that can be applied to the inside perimeter of an apartment. The treatment is diatomaceous earth, a dust made from fossilized remains of diatoms, a type of hard-shelled algae. Researchers said this dust has been used to control pests for more than a century. It clings to the bed bugs as they walk through it, absorbs moisture, and kills them via dehydration.

“We treat the perimeter of the apartment to isolate infestations in one unit and not allow them to spread. It is a lot less expensive to treat one apartment than every unit in the building,” said Dini Miller, a professor of entomology at Virginia Tech.

#SayNOtoPESTICIDES!

Trending today: #Bedbugs are developing a strong resistance to most common insecticides

February 2, 2016 | by Ryan Biek | Newsy

Bedbugs are reportedly building up a strong resistance to some of the most powerful insecticides due to overuse, which means we might need to turn to non-chemical solutions to get rid of them.

Researchers from Virginia Tech and New Mexico State University tested the most common class of insecticide called neonicotinoids, or neonics, which is often combined with pyrethroids in commercial treatments for bedbugs.

Bedbugs are developing a strong resistance to most common insecticides photo

They took a group of bedbugs that came from homes in Ohio and Michigan, which had previously been exposed to neonics, and compared those bedbugs to a population that has been kept in isolation for 30 years, before the insecticide was used.

A third group of bedbugs that was resistant to pyrethroids but never exposed to neonics was also included in the study.

Depending on the specific types of neonic tested, the Ohio and Michigan bedbugs were hundreds to tens of thousands of times more resistant than the isolated group.

The third group’s results were in the middle: more resistant than the isolated group but less resistant than the Ohio and Michigan bedbugs.

Because that third group had never been exposed to neonics, the researchers believe the bedbugs may have a pre-existing resistance mechanism.

The researchers said more non-chemical methods need to be used to combat bedbug infestations. However, they noted the most resistant bedbugs in the study only came from two areas, and not all of the U.S. may be facing this level of resistance.

Rise of the SUPER PESTS: Bed Bugs are resistant to common insecticides. Use of non-chemical methods need to be considered to eradicate Bed Bugs.

  • Scientists tested resistance of four populations to neonicotinoids 
  • They found bugs in Michigan and Cincinnati were resistant to certain types 
  • This means sprays used to kill the bugs aren’t very effective
  • Rise in infestations blamed on travelling, as bugs hitch a ride on clothes

January 28, 2016 | by Sarah Griffiths | MailOnline

They live in the cracks and crevices of beds and crawl out a night to suck blood by detecting our body heat and carbon dioxide.

Now the much loathed bed bug is threatening to become even more of a pest because it is resistant to a common insecticide, scientists warn.

Exotic holidays have been blamed for the recent resurgence of bed bugs in homes as they hitch a ride on clothing or in luggage.

The blood-sucking bed bug (pictured) that's attracted to our body heat and carbon dioxide is threatening to become even more of a pest because it is resistant to a common insecticide, scientists warn.

The blood-sucking bed bug (pictured) that’s attracted to our body heat and carbon dioxide is threatening to become even more of a pest because it is resistant to a common insecticide, scientists warn.

The research has found the parasites have developed a tolerance to neonicotinoids, or neonics, because of their widespread use.

“people are spending a lot of money on products that aren’t working”

It is the first study to show the overuse of certain insecticides has led to an increased resistance to the compounds, making them much less effective than advertised.

In the US alone, millions of dollars are spent on the most widely used commercial chemicals to kill bedbugs, but their overuse has led to an increased resistance to the compounds.

Assistant professor Troy Anderson, from Virginia Tech College of Agriculture and Life Sciences said: ‘While we all want a powerful tool to fight bed bug infestations, what we are using as a chemical intervention is not working as effectively it was designed and, in turn, people are spending a lot of money on products that aren’t working.

New research has found the parasites have developed a tolerance to neonicotinoids, or neonics, because of their widespread use. A stock image of  fumigation is pictured

New research has found the parasites have developed a tolerance to neonicotinoids, or neonics, because of their widespread use. A stock image of fumigation is pictured.

WHERE INFESTATIONS BEGIN

In 2014, genetic tests revealed that a single undetected pregnant bed bug is all it takes to start an entire infestation.

A DNA study at Sheffield University showed colonies of bed bugs come from a common ancestor or a few of the female bed bugs.

The pregnant bed bug could rapidly create a colony of thousands that feed on humans.

Researchers told the BBC that bed bugs’ ability to generate a new colony from such small numbers might be a ‘clue to their recent success’.

‘If you just miss one, they can grow very quickly,’ Professor Roger Butlin said, adding it takes only a few weeks for this to happen.

Bed bugs are capable of surviving without feeding for a month as they wait for a human.

In the late 1880s, an estimated 75 per cent of households were affected, but by the outbreak of World War II, that figure had dwindled to 25 per cent,

Their recent resurgence has been blamed by some experts on resistance to commonly used insecticides and international travel.

‘Unfortunately, the insecticides we were hoping would help solve some of our bed bug problems are no longer as effective as they used to be, so we need to re-evaluate some of our strategies for fighting them.’

Products developed to eradicate infestations in recent years combine both neonics with pyrethroids – another class of insecticide.

Assistant Professor Dr Alvaro Romero from New Mexico State University added: ‘If resistance is detected, products with different modes of action need to be considered, along with the use of non-chemical methods.

‘Companies need to be vigilant for hints of declining performance of products that contain neonicotinoids.

‘For example, bed bugs persisting on previously treated surfaces might be an indication of resistance.

‘In these cases, laboratory confirmation of resistance is advised, and if resistance is detected, products with different modes of action need to be considered, along with the use of non-chemical methods.’

The study, published in the Journal of Medical Entomology, is the first to confirm the resistance.

Researchers collected bed bugs from homes in Cincinnati and Michigan and exposed them to four different neonics: acetamiprid, dinotefuran, imidacloprid and thiamethoxam.

In the US alone, millions of dollars are spent on the most widely used commercial chemicals to kill bedbugs (microscopic image shown) but their overuse has led to an increased resistance to the compounds.

They also used the chemicals on a bed bug colony kept free of insecticide exposure for more than 30 years and to a pyrethroid-resistant population from Jersey City that had not been exposed to neonics since they were collected in 2008.

Those that hadn’t been exposed to the neonics died after contact with very small amounts of the pesticide, while the Jersey City bed bugs showed moderate resistance to acetamiprid and dinotefuran, but not to imidacloprid or thiamethoxam.

The Jersey City colony’s resistance could be due to pre-existing resistance mechanisms.

When exposed to insecticides, bed bugs produce ‘detoxifying enzymes’ to counter them.

Researchers collected bed bugs from homes in Cincinnati and Michigan and exposed them to four different neonics - acetamiprid, dinotefuran, imidacloprid and thiamethoxam. A stock image of fumegation is shown

Researchers collected bed bugs from homes in Cincinnati and Michigan and exposed them to four different neonics – acetamiprid, dinotefuran, imidacloprid and thiamethoxam. A stock image of fumegation is shown

THE CHEMICALS AND BED BUGS

The levels of detoxifying enzymes in the Jersey City bed bugs were higher than those of the susceptible Harlan population.

The Michigan and Cincinnati bed bugs, which were collected after combinations of pyrethroids and neonicotinoids were introduced, had even higher levels of resistance to neonics.

It only took 0.3 nanograms of acetamiprid to kill 50% of the non-resistant bed bugs from Dr Harlan’s lab, but it took more than 10,000 nanograms to kill 50% of the Michigan and Cincinnati bed bugs.

Just 2.3 nanograms of imidacloprid was enough to kill 50% t of the Harlan bed bugs, but it took 1,064 and 365 nanograms to kill the Michigan and Cincinnati bed bugs.

The numbers were similar for dinotefuran and thiamethoxam.

Compared to the Harlan control group, the Michigan bed bugs were 462 times more resistant to imidacloprid, 198 times more resistant to dinotefuran, 546 times more resistant to thiamethoxam, and 33,333 times more resistant to acetamiprid.

The Cincinnati bed bugs were 163 times more resistant to imidacloprid, 226 times more resistant to thiamethoxam, 358 times more resistant to dinotefuran, and 33,333 times more resistant to acetamiprid.

The levels of detoxifying enzymes in the Jersey City bed bugs were higher than those of the susceptible Harlan population.

Professor Romero explained: ‘Elevated levels of detoxifying enzymes induced by other classes of insecticides might affect the performance of newer insecticides.’

The Michigan and Cincinnati bed bugs, which were collected after combinations of pyrethroids and neonicotinoids were introduced, had even higher levels of resistance to neonics.

It only took 0.3 nanograms of acetamiprid to kill 50 per cent of the non-resistant bed bugs from Dr Harlan’s lab, but it took more than 10,000 nanograms to kill 50 per cent of the Michigan and Cincinnati bed bugs.

Just 2.3 nanograms of imidacloprid was enough to kill 50 per cent of the Harlan bed bugs, but it took 1,064 and 365 nanograms to kill the Michigan and Cincinnati bed bugs, respectively.

The numbers were similar for dinotefuran and thiamethoxam.

Compared to the Harlan control group, the Michigan bed bugs were 462 times more resistant to imidacloprid, 198 times more resistant to dinotefuran, 546 times more resistant to thiamethoxam, and 33,333 times more resistant to acetamiprid.

The Cincinnati bed bugs were 163 times more resistant to imidacloprid, 226 times more resistant to thiamethoxam, 358 times more resistant to dinotefuran, and 33,333 times more resistant to acetamiprid.

SayNOtoPESTICIDES!

Heartbreaking and True…..Woman who started blaze trying to kill bed bugs: ‘I am so sorry’

– November 5, 2015

The woman who started a massive fire at an apartment building in Detroit is speaking exclusively to FOX 2.

Distraught and desperate for forgiveness, the woman believed to be responsible for the fire at the Grayton Park Apartments on the city’s west side spoke with FOX 2 by phone from Sinai Grace Hospital. She says she was trying to get rid of the bed bugs that had been in her apartment for more than a year.

“They had sent exterminators over, but it didn’t work, and these things just kept at me, they just kept at me,” she says. “I went in there with clear skin, now my skin is all bitten up and looking like leather. I just want to get rid of these bed bug tormentors.”

The woman, who wished to remain anonymous, says she killed some of the bedbugs a few weeks ago by turning up the heat in her west side apartment, which is near Outer Drive and Rouge Park. So, she says she did the same Tuesday and doused her floors with alcohol, which is often used to kill bedbugs. But, she also had her oven on and she did not know the fumes are flammable.

“When I was standing in the front room, the whole floor just ignited. The whole floor just ignited! I couldn’t get the fire extinguisher,” she says.

The flames spread throughout the building and the roof collapsed on firefighters. Three of them taken to the hospital, and more than two dozen families were burned out of their homes.

For the woman who says she mistakenly started the fire, the weight of that reality is almost overwhelming.

“I’m so sorry, I’m so sorry. If I could, I would take it all back,” she says. “Can you please ask them to forgive me? I didn’t mean to set the fire, okay? You can tell everybody.”

The woman says she tried to get her neighbors out of the building and thankfully no one died.

The fire department is still investigating what happened, they are not saying what started the fire. It is unclear if the woman will face any charges.